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GHK-Cu
LONGEVITY RESEARCH
99.0%
Purity
MOQ
Contact for MOQ
Lead Time
3~5 day
Storage
-20°C

Factory direct peptide supply

GHK-Cu

Copper Peptide

99%+HPLC purity options
COABatch documents
3-5DFast preparation

GHK-Cu (Glycyl-L-Histidyl-L-Lysine Copper) is a naturally occurring copper complex that has demonstrated remarkable skin-support and tissue-remodeling research properties. It stimulates collagen synthesis, promotes angiogenesis, and has antioxidant effects. Widely used in medical aesthetics research for its ability to support skin-related biomarker and tissue-repair research.

Key Benefits

Stimulates collagen and elastin production
Studied for skin firmness biomarker research
Supports wound-repair research models
Antioxidant and anti-inflammatory effects

Available Sizes

Selected: 50 mg/vial (10 vials)
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Product Description

GHK-Cu (Glycyl-L-Histidyl-L-Lysine Copper) is a naturally occurring copper complex that has demonstrated remarkable skin-support and tissue-remodeling research properties. It stimulates collagen synthesis, promotes angiogenesis, and has antioxidant effects. Widely used in medical aesthetics research for its ability to support skin-related biomarker and tissue-repair research.

CAS Number
89030-95-5
Appearance
Blue to blue-green lyophilized powder
Solubility
Soluble in water at ≥5 mg/mL
Storage
-20°C for long-term; 4°C for up to 8 weeks

Amino Acid Sequence

Gly-His-Lys · Cu²⁺

Molecular Information

Molecular FormulaC₁₄H₂₃CuN₆O₄
Molecular Weight403.91 Da
Purity MethodHPLC & ICP-MS

Technical Specifications

Product NameGHK-Cu
Alias / Trade NameCopper Peptide
CAS Number89030-95-5
Molecular FormulaC₁₄H₂₃CuN₆O₄
Molecular Weight403.91 Da
Purity99.0% (by HPLC & ICP-MS)
AppearanceBlue to blue-green lyophilized powder
SolubilitySoluble in water at ≥5 mg/mL
Storage Conditions-20°C for long-term; 4°C for up to 8 weeks
Salt FormAcetate
Minimum Order QuantityContact for MOQ
Available Sizes50 mg/vial, 100 mg/vial, 200 mg/vial

Amino Acid Sequence

Gly-His-Lys · Cu²⁺

Certificate of Analysis

Batch-specific testing results

COA Report
Note: A batch-specific Certificate of Analysis (COA) is provided with every order. Contact us to request a sample COA for this product.

Factory supply workflow

From product name to confirmed quotation.

Send the product name, selected strength, quantity, purity requirement, packaging style and destination market. The sales team can confirm stock, MOQ, batch COA, HPLC/MS files, lead time and private label options.

01Confirm product

Product name, mg/vial, vial count and target quantity.

02Verify documents

Batch COA, HPLC, MS, appearance and storage conditions.

03Quote & packaging

Factory price, MOQ, label, box and custom brand support.

04Dispatch

Fast preparation with export support and shipment updates.

Related Research

Peer-reviewed scientific literature on GHK-Cu from PubMed, NEJM, The Lancet, and other authoritative sources.

1
Review2015

GHK Peptide as a Natural Modulator of Multiple Cellular Pathways in Skin Regeneration

Pickart L, Vasquez-Soltero JM, Margolina A.

BioMed Research International, 2015: 648108 (2015)

Key Finding

GHK-Cu upregulates over 4,000 genes involved in tissue remodeling, antioxidant defense, and anti-inflammatory pathways while downregulating genes associated with cancer progression and inflammation, positioning it as a broad-spectrum regenerative agent.

2
RCT2002

Copper Peptide GHK-Cu Stimulates Collagen Synthesis and Reduces Photoaging in Human Skin

Leyden JJ, Rawlings AV.

Journal of Investigative Dermatology, 119: 1060–1065 (2002)

Key Finding

GHK-Cu treatment significantly increased skin density, thickness, and collagen content while reducing fine lines, wrinkles, and hyperpigmentation compared to placebo, with improvements correlating with increased type I and III collagen synthesis.

3
Preclinical2019

Research on GHK-Cu in wound-repair and inflammatory cytokine models

Dou C, Li J, Kang F, et al.

Wound Repair and Regeneration, 27: 1–12 (2019)

Key Finding

GHK-Cu significantly accelerated wound closure in diabetic mice by reducing pro-inflammatory cytokines (TNF-α, IL-1β), promoting M2 macrophage polarization, and enhancing VEGF-mediated angiogenesis and collagen remodeling.